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Patients reported symptomatic improvement in OAB even among participants who did not experience significant weight loss during GLP-1 receptor agonist therapy, suggesting that the observed benefit may be independent of weight reduction [36]

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Two possible mechanisms of NO-mediated protein S-glutathionylation can be envisioned: through a GSNO (activated thiol) formation and its consequent reaction with protein-thiol (179) or through an intermediate protein-thiol nitrosylation (activated protein-thiol: analog of sulfenic acid) and its consequent reaction with GSH

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